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- Supraphysiologic doses of anabolic-androgenic steroids , which embody greater than 30 natural and artificial derivatives of testosterone, could cause a large spectrum of medical sequalae.
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Moreover, testosterone has been used in the treatment of decreased muscle mass and bone density as properly as anemia. The position testosterone may play in the improvement of cardiovascular disease has more and more turn out to be the subject of current curiosity . Anabolic androgenic steroid use has elevated over the past decade, despite its potential for opposed results. They continue for use by Olympic level athletes, and have even spread to high school level athletes, noncompetitive bodybuilders and leisure athletes (Sullivan et al 1998; Dhar et al 2007).
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Anabolic Steroids, Acute Myocardial Infarction And Polycythemia: A Case Report And Review Of The Literature
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Unfortunately, the information on the utilization of excessive dose anabolic steroids in people are primarily offered as case reports or small studies that lack enough management teams. In addition, the data out there within the literature does not account for the steroid kind or dose, as neither may be recognized. Patients also may be taking concomitant stimulants, such as ephedra, which can confound knowledge and the potential for opposed occasions. It is in all probability going that the frequency of cardiac events is underreported in the medical literature. Supraphysiologic doses of anabolic-androgenic steroids , which embrace more than 30 natural and artificial derivatives of testosterone, could trigger a large spectrum of medical sequalae.
We report a case of an acute ST-segment elevation myocardial infarction in a patient with traditional cardiac risk factors using supraphysiologic doses of supplemental, intramuscular testosterone. In addition, this affected person also had polycythemia, doubtless secondary to high-dose testosterone. The patient underwent profitable percutaneous intervention of the best coronary artery. Physicians and patients should be aware of the clinical penalties of anabolic steroid abuse. Development of an atheromatous plaque perpetuates endothelial dysfunction and promotes platelet aggregation and intracoronary thrombus formation (Ajayi et al 1995; Nieminen et al 1996). Moreover, a part of endothelial dysfunction has been proposed, which can contribute to abnormal vessel reactivity.
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However, testosterone in physiologic doses may even be beneficial in patients with angina , have larger ischemic thresholds and improved quality of life . Short-term intracoronary administration of testosterone induces coronary artery dilation and increases coronary blood move in males with established coronary artery illness . Therefore, an interaction of endogenous elements of the patient’s danger profile as well as the testosterone dosing may be issues of whether antagonistic occasions occur. Erythrocytosis has been reported as a potential impact of testosterone, which may be helpful in patients with anemia. The biggest threat of erythrocytosis occurs with injectable types of testosterone compared with transdermal preparations, particularly in supraphysiologic doses. An improve of as much as 44% of the baseline hematocrit has been reported .
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